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Core designing of a new type of TVS-2M FAs: neutronics and thermal-hydraulics design basis limits

Saeed GHAEMI, Farshad FAGHIHI

《能源前沿（英文）》 2021年第15卷第1期页码 256-278 doi: 10.1007/s11708-018-0583-x

摘要： One of the most important aims of this study is to improve the core of the current VVER reactors to achieve more burn-up (or more cycle length) and more intrinsic safety. It is an independent study on the Russian new proposed FAs, called TVS-2M, which would be applied for the future advanced VVERs. Some important aspects of neutronics as well as thermal hydraulics investigations (and analysis) of the new type of Fas are conducted, and results are compared with the standards PWR CDBL. The TVS-2M FA contains gadolinium-oxide which is mixed with UO (for different Gd densities and U-235 enrichments which are given herein), but the core does not contain BARs. The new type TVS-2M Fas are modeled by the SARCS software package to find the PMAXS format for three states of CZP and HZP as well as HFP, and then the whole core is simulated by the PARCS code to investigate transient conditions. In addition, the WIMS-D5 code is suggested for steady core modeling including TVS-2M FAs and/or TVS FAs. Many neutronics aspects such as the first cycle length (first cycle burn up in terms of MW d/kgU), the critical concentration of boric acid at the BOC as well as the cycle length, the axial, and radial power peaking factors, differential and integral worthy of the most reactive CPS-CRs, reactivity coefficients of the fuel, moderator, boric acid, and the under-moderation estimation of the core are conducted and benchmarked with the PWR CDBL. Specifically, the burn-up calculations indicate that the 45.6 d increase of the first cycle length (which corresponds to 1.18 MW d/kgU increase of burn-up) is the best improving aim of the new FA type called TVS-2M. Moreover, thermal-hydraulics core design criteria such as MDNBR (based on W3 correlation) and the maximum of fuel and clad temperatures (radially and axially), are investigated, and discussed based on the CDBL.

关键词： TVS-2M FAs core design basis limits VVER-1000 analysis mixture of uranium-gadolinium oxides fuels thermal-hydraulics PARCS WIMS-D5

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WIMS/CITATION在IHNI-1堆芯物理计算中的应用

赵柱民,张良,江新标,陈立新,朱养妮,周永茂

《中国工程科学》 2012年第14卷第8期页码 38-41

摘要：

建立了利用WIMS＋CITATION计算医院中子照射器I型堆堆芯中子学参数的模型，计算了堆芯的功率分布、顶铍反应性价值、控制棒价值、温度系数

关键词：医院中子照射器I型堆堆芯中子 WIMS CITATION

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Keratin 5-Cre-driven deletion of

Jun Yang, Lianqing Wang, Yingzhi Huang, Keqiang Liu, Chaoxia Lu, Nuo Si, Rongrong Wang, Yaping Liu, Xue Zhang

《医学前沿（英文）》 2020年第14卷第3期页码 305-317 doi: 10.1007/s11684-019-0722-8

摘要： Familial acne inversa (AI) is an autoinflammatory disorder that affects hair follicles and is caused by loss-of-function mutations in -secretase component genes. We and other researchers showed that ( ) is the most frequently mutated gene in familial AI. In this study, we generated a keratin 5-Cre-driven epidermis-specific conditional knockout mutant in mice. We determined that this mutant recapitulated the major phenotypes of AI, including hyperkeratosis of hair follicles and inflammation. In mice, the IL-36a expression level markedly increased starting from postnatal day 0 (P0), and this increase occurred much earlier than those of TNF- , IL-23A, IL-1 and TLR4. RNA-Seq analysis indicated that Sprr2d, a member of the small proline-rich protein 2 family, in the skin tissues of the mice was also upregulated on P0. Quantitative reverse-transcription polymerase chain reaction showed that other genes had a similar expression pattern. Our findings suggested that IL-36a might be a key inflammatory cytokine in the pathophysiology of AI and involved in the malfunction of the skin barrier in the pathogenesis of AI.

关键词： acne inversa mouse model interleukin 1 family member 6 small proline rich protein 2D key inflammatory cytokine

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基于WIMS和MCNP耦合程序的医院中子照射器I型堆燃耗计算

郭和伟,江新标,赵柱民,陈立新,张信一,周永茂

《中国工程科学》 2012年第14卷第8期页码 64-68

摘要：

建立了基于WIMS和MCNP的临界-燃耗耦合计算方法，并对此方法进行了验算。

关键词：医院中子照射器I型堆耦合程序燃耗

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5G成效未达预期

Mitch Leslie

《工程（英文）》 2023年第29卷第10期页码 5-7 doi: 10.1016/j.eng.2023.08.003

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基于 5G技术的教育信息化应用研究

卢向群,孙禹

《中国工程科学》 2019年第21卷第6期页码 120-128 doi: 10.15302/J-SSCAE-2019.06.020

摘要：

教育信息化日益普及，智慧学习空间不断出现，学生对学习内容的个性化、便捷化、多样化需求也日益凸显，而第五代移动通信（ 5G）技术以其大带宽、本文重点探索 5G技术的教育信息化应用，在梳理教育信息化主要特征的基础上，分析了我国教育网络面临的技术挑战，从教学、教研、教育管理三方面着手，论证提出相应的 5G应用场景，形成 5G教育信息化总体视图。面向 2035年， 5G的技术普及与应用拓展，将不断丰富教育场景的内涵，推动教育场景的变革，成为我国教育信息化创新的原生动力。

关键词：教育信息技术教育信息化 5G 技术应用场景

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5G应用下的MIMO阵列天线去耦方法综述 Review Articles

Xiao-xi ZHANG, Ai-di REN, Ying LIU

《信息与电子工程前沿（英文）》 2020年第21卷第1期页码 62-71 doi: 10.1631/FITEE.1900466

摘要：提出一种面向微纳卫星的自主汽化管理液氨推进系统。相比常规冷气或液化气推进系统，提出多路平行筛孔式汽化装置和对应汽化控制方法。所提方案有效解决了液氨高汽化潜热和不易完全汽化的问题，从而发挥液氨推进剂高贮存密度和高比冲性能优势。此外，重点分析自主汽化管理液氨推进系统的工作流程及其涉及的物理化学过程和数学模型。综合考虑推力表现和能源效率，提出最优系统推力控制策略。地面测试表明，自主汽化管理液氨推进系统总重1.8 kg（包含0.34 kg液氨推进剂），比冲达到100 s，系统功耗在10 W以下。自主汽化管理液氨推进系统具有高比冲、低功耗、可实现性强、推力输出均一稳定等特点，适合微纳卫星在轨应用。

关键词： MIMO阵列；5G手机；去耦方法

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lncR-GAS5 upregulates the splicing factor to impair endothelial autophagy, leading to atherogenesis

《医学前沿（英文）》 2023年第17卷第2期页码 317-329 doi: 10.1007/s11684-022-0931-4

摘要： Long noncoding RNAs (lncRNAs) play a critical role in the regulation of atherosclerosis. Here, we investigated the role of the lncRNA growth arrest-specific 5 (lncR-GAS5) in atherogenesis. We found that the enforced expression of lncR-GAS5 contributed to the development of atherosclerosis, which presented as increased plaque size and reduced collagen content. Moreover, impaired autophagy was observed, as shown by a decreased LC3II/LC3I protein ratio and an elevated P62 level in lncR-GAS5-overexpressing human aortic endothelial cells. By contrast, lncR-GAS5 knockdown promoted autophagy. Moreover, serine/arginine-rich splicing factor 10 (SRSF10) knockdown increased the LC3II/LC3I ratio and decreased the P62 level, thus enhancing the formation of autophagic vacuoles, autolysosomes, and autophagosomes. Mechanistically, lncR-GAS5 regulated the downstream splicing factor SRSF10 to impair autophagy in the endothelium, which was reversed by the knockdown of SRSF10. Further results revealed that overexpression of the lncR-GAS5-targeted gene miR-193-5p promoted autophagy and autophagic vacuole accumulation by repressing its direct target gene, SRSF10. Notably, miR-193-5p overexpression decreased plaque size and increased collagen content. Altogether, these findings demonstrate that lncR-GAS5 partially contributes to atherogenesis and plaque instability by impairing endothelial autophagy. In conclusion, lncR-GAS5 overexpression arrested endothelial autophagy through the miR-193-5p/SRSF10 signaling pathway. Thus, miR-193-5p/SRSF10 may serve as a novel treatment target for atherosclerosis.

关键词： lncR-GAS5 miR-193-5p splicing factor SRSF10 autophagy atherogenesis

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2020普遍通信与5G技术国际会议(UC5G 2020)

会议日期： 2020年03月26日

会议地点：上海

主办单位： APISE

Boehmite-supported CuO as a catalyst for catalytic transfer hydrogenation of 5-hydroxymethylfurfural

《化学科学与工程前沿（英文）》 2023年第17卷第4期页码 415-424 doi: 10.1007/s11705-022-2225-4

摘要： 2,5-bis(hydroxymethyl)furan (BHMF) is an important monomer of polyester. Its oxygen-containing rigid ring structure and symmetrical diol functional group establish it as an alternative to petroleum-based monomer with unique advantages for the prodution of the degradable bio-based polyester materials. Herein, we prepared a boehmite-supported copper-oxide catalyst for the selective hydrogenation of 5-hydroxymethylfurfural into BHMF via catalytic transfer hydrogenation (CTH). Further, ethanol successfully replaced conventional high-pressure hydrogen as the hydrogen donor, with up to 96.9% BHMF selectivity achieved under suitable conditions. Through characterization and factor investigations, it was noted that CuO is crucial for high BHMF selectivity. Furthermore, kinetic studies revealed a higher by-product activation energy compared to that of BHMF, which explained the influence of reaction temperature on product distribution. To establish the catalyst structure-activity correlation, a possible mechanism was proposed. The copper-oxide catalyst deactivated following CTH because ethanol reduced the CuO, which consequently decreased the active sites. Finally, calcination of the catalyst in air recovered its activity. These results will have a positive impact on hydrogenation processes in the biomass industry.

关键词： biomass 5-hydroxymethylfurfural 2 5-bis(hydroxymethyl)furan transfer hydrogenation catalysis

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一场潜在的5G干扰风暴

Chris Palmer

《工程（英文）》 2019年第5卷第5期页码 815-816 doi: 10.1016/j.eng.2019.08.007

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2020年5G移动通信与信息科学国际学术会议（MCIS-5G2020）

会议日期： 2020年06月26日

会议地点：广东广州

5G综述及中国的进展 None

Shan-zhi CHEN, Shao-li KANG

《信息与电子工程前沿（英文）》 2018年第19卷第3期页码 309-321 doi: 10.1631/FITEE.1800070

摘要： 5G技术、标准和产业化从20012年开始高速发展，是全球经济新的驱动力。本文全面介绍了5G，包括愿景、要求、演进、关键技术、标准化、频谱分配、技术试验、产业化进展和5G主要贡献者。同时指出，中国企业作为5G主要技术和标准的贡献者，正处于“5G引领”过程中。最后介绍了5G最新进展和作者关于5G的再思考。

关键词： 5G；IMT-2020；关键技术；标准；现场试验

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Management innovation of Chang’e-5 project

《工程管理前沿（英文）》 2021年第8卷第4期页码 620-626 doi: 10.1007/s42524-021-0165-1

关键词： lunar exploration sample return systems engineering project management

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Sodium butyrate activates HMGCS2 to promote ketone body production through SIRT5-mediated desuccinylation

《医学前沿（英文）》 2023年第17卷第2期页码 339-351 doi: 10.1007/s11684-022-0943-0

摘要： Ketone bodies have beneficial metabolic activities, and the induction of plasma ketone bodies is a health promotion strategy. Dietary supplementation of sodium butyrate (SB) is an effective approach in the induction of plasma ketone bodies. However, the cellular and molecular mechanisms are unknown. In this study, SB was found to enhance the catalytic activity of 3-hydroxy-3-methylglutaryl-CoA synthase 2 (HMGCS2), a rate-limiting enzyme in ketogenesis, to promote ketone body production in hepatocytes. SB administrated by gavage or intraperitoneal injection significantly induced blood β-hydroxybutyrate (BHB) in mice. BHB production was induced in the primary hepatocytes by SB. Protein succinylation was altered by SB in the liver tissues with down-regulation in 58 proteins and up-regulation in 26 proteins in the proteomics analysis. However, the alteration was mostly observed in mitochondrial proteins with 41% down- and 65% up-regulation, respectively. Succinylation status of HMGCS2 protein was altered by a reduction at two sites (K221 and K358) without a change in the protein level. The SB effect was significantly reduced by a SIRT5 inhibitor and in Sirt5-KO mice. The data suggests that SB activated HMGCS2 through SIRT5-mediated desuccinylation for ketone body production by the liver. The effect was not associated with an elevation in NAD⁺/NADH ratio according to our metabolomics analysis. The data provide a novel molecular mechanism for SB activity in the induction of ketone body production.

关键词： sodium butyrate succinylation HMGCS2 ketogenesis SIRT5